A:
The issue is that the path does not exist. This occurs when there is an entry in your hosts file for 127.0.0.1 (because you use the loopback interface) and it points to a path that is a symlink or directory. You can check the existence of the symlink by listing it like this:
for f in `ls -l /path/to/symlink`
do
if test -e "$f"; then
echo "Exists!"
else
echo "Does not exist"
fi
done
In your case it is the path to the directory that is causing the problem.
The easiest way to fix it would be to remove the entry in the hosts file. I would also recommend you check for any similar entries in your hosts file, otherwise you may end up with the same problem at a later date.
Insulin and insulin-like growth factors stimulate glucose uptake in human adipocytes through phosphatidylinositol 3-kinase and phosphatidylinositol 3,4,5-trisphosphate-dependent signal transduction mechanisms.
Recent studies have indicated that insulin and insulin-like growth factors can stimulate glucose uptake into human adipocytes. The purpose of this study was to further examine the signal transduction mechanisms by which insulin and insulin-like growth factors stimulate glucose uptake in human adipocytes. Insulin stimulated an increase in 3-O-methylglucose uptake into isolated human adipocytes. The effect of insulin on 3-O-methylglucose uptake was dose dependent (half-maximal stimulation at approximately 0.5 nmol/L) and was time dependent (maximal stimulation at approximately 5 min). The ability of insulin to stimulate 3-O-methylglucose uptake was not inhibited by inhibitors of protein kinase C, phospholipase C, or phospholipase A2. Insulin stimulation of 3-O-methylglucose uptake was partially inhibited by the lipid-depleting agent, 5-dodecyl-6-D-glucose. In contrast, insulin-like growth factor I (IGF-I) stimulated glucose uptake and potentiated the action of insulin on glucose uptake. Insulin and IGF-I stimulated increases in intracellular calcium concentration in human adipocytes. be359ba680
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